Pseudo Pemphigus Phenotypes in Mice with Inactivated Desmoglein 3

Author:

Grando Sergei A.,Pittelkow Mark R.

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference13 articles.

1. A spontaneous deletion within the desmoglein 3 extracellular domain of mice results in hypomorphic protein expression, immunodeficiency, and a wasting disease phenotype;Kountikov;Am J Pathol,2015

2. Sundberg JP, Shultz LD, King LE, Montagutelli X: The Spontaneous Balding and Desmoglein 3 Null Mutations: Mouse Models for Pemphigus Vulgaris. JAX NOTES [Internet], 1998. Available at http://jaxmice.jax.org/jaxnotes/archive/473.pdf

3. Mice expressing a mutant desmosomal cadherin exhibit abnormalities in desmosomes, proliferation, and epidermal differentiation;Allen;J Cell Biol,1996

4. A mouse model of pemphigus vulgaris by adoptive transfer of naive splenocytes from desmoglein 3 knockout mice;Aoki-Ota;Br J Dermatol,2004

5. Involvement of a tissue-specific autoantibody in skin disorders of murine systemic lupus erythematosus and autoinflammatory diseases;Nishimura;Proc Natl Acad Sci U S A,2006

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1. In Vitro, Ex Vivo, and In Vivo Models for the Study of Pemphigus;International Journal of Molecular Sciences;2022-06-24

2. Current status and prospects for the diagnosis of pemphigus vulgaris;Expert Review of Clinical Immunology;2021-07-06

3. Role of Intravenous Immunoglobulin in Dermatologic Disorders;Biologic and Systemic Agents in Dermatology;2017-10-03

4. Monopathogenic vs multipathogenic explanations of pemphigus pathophysiology;Experimental Dermatology;2016-10-28

5. Authors' Reply;The American Journal of Pathology;2015-11

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