Liver-Specific, but Not Retina-Specific, Hepcidin Knockout Causes Retinal Iron Accumulation and Degeneration-Reference-Cited by-同舟云学术

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Liver-Specific, but Not Retina-Specific, Hepcidin Knockout Causes Retinal Iron Accumulation and Degeneration

Published:2019-09 Issue:9 Volume:189 Page:1814-1830
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Baumann Bailey H.,Shu Wanting,Song Ying,Sterling Jacob,Kozmik Zbynek,Lakhal-Littleton Samira,Dunaief Joshua L.

Funder

National Eye Institute

UPenn

Research to Prevent Blindness

F.M. Kirby Foundation

Paul and Evanina Bell Mackall Foundation Trust

MEYS

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference51 articles.

1. Iron biology in immune function, muscle metabolism and neuronal functioning;Beard;J Nutr,2001

2. Chemistry and biology of eukaryotic iron metabolism;Aisen;Int J Biochem Cell Biol,2001

3. Differential sensitivity of cones to iron-mediated oxidative damage;Rogers;Invest Ophthalmol Vis Sci,2007

4. Maculas affected by age-related macular degeneration contain increased chelatable iron in the retinal pigment epithelium and Bruch's membrane;Hahn;Arch Ophthalmol,2003

5. Iron metabolism in the CNS: implications for neurodegenerative diseases;Rouault;Nat Rev Neurosci,2013

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