Notch3 Signaling and Aggregation as Targets for the Treatment of CADASIL and Other NOTCH3-Associated Small-Vessel Diseases-Reference-Cited by-同舟云学术

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Notch3 Signaling and Aggregation as Targets for the Treatment of CADASIL and Other NOTCH3-Associated Small-Vessel Diseases

Published:2021-11 Issue:11 Volume:191 Page:1856-1870
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Schoemaker Dorothee^ORCID,Arboleda-Velasquez Joseph F.

Funder

National Institutes of Health

National Institute on Aging

National Institute of Neurological Disorders and Stroke

American Heart Association

Fonds de Recherche du Québec - Santé

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference101 articles.

1. Cadasil;Chabriat;Lancet Neurol,2009

2. Notch3 mutations in CADASIL, a hereditary adult-onset condition causing stroke and dementia;Joutel;Nature,1996

3. Long-term prognosis and causes of death in CADASIL: a retrospective study in 411 patients;Opherk;Brain,2004

4. CADASIL: treatment and management options;Bersano;Curr Treat Options Neurol,2017

5. Notch3 is required for arterial identity and maturation of vascular smooth muscle cells;Domenga;Genes Dev,2004

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2. Stroke genetics and how it Informs novel drug discovery;Expert Opinion on Drug Discovery;2024-03-04

3. Protein aggregates containing wild-type and mutant NOTCH3 are major drivers of arterial pathology in CADASIL;Journal of Clinical Investigation;2024-02-22

4. Distinctive feeding vessel architecture and sparse collateralization may underlie the characteristic early temporal pole and external capsule white matter compromise in CADASIL;Medical Hypotheses;2024-02

5. Epidermal Growth Factor Receptors in Vascular Endothelial Cells Contribute to Functional Hyperemia in the Brain;International Journal of Molecular Sciences;2023-11-14

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