Abnormal regulation of mammalian p21ras contributes to malignant tumor growth in von Recklinghausen (type 1) neurofibromatosis-Reference-Cited by-同舟云学术

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Abnormal regulation of mammalian p21ras contributes to malignant tumor growth in von Recklinghausen (type 1) neurofibromatosis

Published:1992-04 Issue:2 Volume:69 Page:265-273
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

DeClue Jeffrey E.,Papageorge Alex G.,Fletcher Jonathan A.,Diehl Scott R.,Ratner Nancy,Vass William C.,Lowy Douglas R.

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference58 articles.

1. Guanosine triphosphatase activating protein (GAP) interacts with the p21 ras effector binding domain;Adari;Science,1988

2. Genetic analysis of mammalian GAP expressed in yeast;Ballester;Cell,1989

3. The NF1 locus encodes a protein functionally related to mammalian GAP and yeast IRA proteins;Ballester;Cell,1990

4. ras genes;Barbacid;Annu. Rev. Biochem.,1987

5. Differential regulation of ras GAP and neurofibromatosis gene product activities;Bollag;Nature,1991

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