Mitochondrial degradation of amyloidogenic proteins — A new perspective for neurodegenerative diseases-Reference-Cited by-同舟云学术

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Mitochondrial degradation of amyloidogenic proteins — A new perspective for neurodegenerative diseases

Published:2019-10 Issue: Volume:181 Page:101660
ISSN:0301-0082
Container-title:Progress in Neurobiology
language:en
Short-container-title:Progress in Neurobiology

Author:

Lautenschläger Janin,Kaminski Schierle Gabriele S.

Funder

Deutsche Forschungsgemeinschaft

Wellcome Trust

UK Medical Research Council (MRC)

Alzheimer Research UK (ARUK)

Infinitus China Ltd

Publisher

Elsevier BV

Subject

General Neuroscience

Reference93 articles.

1. Decreased proteolytic activity of the mitochondrial amyloid-β degrading enzyme, PreP peptidasome, in alzheimer’s disease brain mitochondria;Alikhani;J. Alzheimers Dis.,2011

2. Mitochondrial targeting and a novel transmembrane arrest of Alzheimer’s amyloid precursor protein impairs mitochondrial function in neuronal cells;Anandatheerthavarada;J. Cell Biol.,2003

3. Environmental risk factors and Parkinson’s disease: an umbrella review of meta-analyses;Bellou;Parkinsonism Relat. Disord.,2016

4. TOM40 mediates mitochondrial dysfunction induced by α-synuclein accumulation in Parkinson’s disease;Bender;PLoS One,2013

5. LON is the master protease that protects against protein aggregation in human mitochondria through direct degradation of misfolded proteins;Bezawork-Geleta;Sci. Rep.,2015

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