Diamide acts intracellularly to enhance transmitter release: The differential permeation of diamide, DIP, DIP + 1 and DIP + 2 across the nerve terminal membrane

Author:

Carlen P.L.,Kosower E.M.,Werman R.

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference14 articles.

1. The ‘desensitizing’ effect of acetylcholine on the mammalian motor end-plate;Axelsson;Acta physiol. scand.,1958

2. Facilitation of the frog neuromuscular junction during and after repetitive stimulation;Braun;Pflu¨gers Arch. ges. Physiol.,1966

3. The thiol-oxidizing agent diamide increases transmitter release by decreasing calcium requirements for neuromuscular transmission in the frog;Carlen;Brain Research,1976

4. The effect of acetylcholine on neuromuscular transmission in the frog;Ciani;J. Pharmacol. exp. Ther.,1963

5. The effect of acetylcholine upon mammalian motor nerve terminals;Hubbard;J. Physiol. (Lond.),1965

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