The thiol-oxidizing agent diamide increases transmitter release by decreasing calcium requirements for neuromuscular transmission in the frog
Author:
Publisher
Elsevier BV
Subject
Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience
Reference62 articles.
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4. The role of a reacting disulphide bond in the function of the acetylcholine receptor at the frog neuromuscular junction;Ben-Haim;J. Physiol. (Lond.),1973
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1. Modification of ionic currents underlying action potentials in mouse nerve terminals by the thioloxidizing agent diamide;Neuropharmacology;1995-11
2. [11] Diamide: An oxidant probe for thiols;Methods in Enzymology;1995
3. Sensory neuropeptide-mediated bronchoconstriction of the guinea pig lung by diamide; a comparison to hydrogen peroxide;European Journal of Pharmacology: Environmental Toxicology and Pharmacology;1994-04
4. Effect of the thiol-oxidizing agent diamide on NH2Cl-induced rat colonic electrolyte secretion;American Journal of Physiology-Cell Physiology;1993-07-01
5. Reversal by cysteine of the cadmium-induced block of skeletal neuromuscular transmission in vitro;British Journal of Pharmacology;1992-09
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