Changes in Ca2+-dependent protein kinase C isoforms induced by chronic ethanol treatment in mice-Reference-Cited by-同舟云学术

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Changes in Ca2+-dependent protein kinase C isoforms induced by chronic ethanol treatment in mice

Published:2001-07 Issue:2 Volume:307 Page:85-88
ISSN:0304-3940
Container-title:Neuroscience Letters
language:en
Short-container-title:Neuroscience Letters

Author:

Narita Minoru,Tamaki Hiroko,Kobayashi Misato,Soma Miho,Narita Michiko,Suzuki Tsutomu

Publisher

Elsevier BV

Subject

General Neuroscience

Reference18 articles.

1. Acute and chronic ethanol treatments alter GABA receptor-operated chloride channels;Allan;Pharmacol. Biochem. Behav.,1987

2. Activation of cyclic AMP-dependent protein kinase reverses tolerance of a nucleotide transporter to ethanol;Coe;J. Phamacol. Exp. Ther.,1996

3. Ethanol exposure increases total protein kinase C activity in human lymphocytes;DePetrillo;Alcohol. Clin. Exp. Res.,1993

4. Pharmacological aspects of physical dependence on ethanol;Goldstein;Life Sci.,1976

5. Cellular and behavioral neurobiology of alcohol;Grant;Clin. Neurosci.,1995

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1. Differential regulation of G protein‐coupled receptor‐associated proteins in the caudate and the putamen of cynomolgus macaques following chronic ethanol drinking;Journal of Neurochemistry;2024-05-23

2. Alteration of orexin-A and PKCα in the postmortem brain of pure-opioid and multi-drug abusers;Neuropeptides;2020-10

3. Chronic alcohol feeding potentiates hormone-induced calcium signalling in hepatocytes;The Journal of Physiology;2017-04-18

4. Changes in the expression of transthyretin and protein kinase Cγ genes in the prefrontal cortex in response to naltrexone;Neuroscience Letters;2011-01

5. Protein kinase C isozymes as regulators of sensitivity to and self-administration of drugs of abuse-studies with genetically modified mice;Behavioural Pharmacology;2010-09

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