Biochemical and Objective Response to Abiraterone Acetate Withdrawal: Incidence and Clinical Relevance of a New Scenario for Castration-resistant Prostate Cancer
Author:
Publisher
Elsevier BV
Subject
Urology
Reference27 articles.
1. The 16,17-double bond is needed for irreversible inhibition of human cytochrome p45017alpha by abiraterone (17-(3-pyridyl)androsta-5, 16-dien-3beta-ol) and related steroidal inhibitors;Jarman;J Med Chem,1998
2. Selective inhibition of CYP17 with abiraterone acetate is highly active in the treatment of castration-resistant prostate cancer;Attard;J Clin Oncol,2009
3. Significant and sustained antitumor activity in post-docetaxel, castration-resistant prostate cancer with the CYP17 inhibitor abiraterone acetate;Reid;J Clin Oncol,2010
4. Phase II multicenter study of abiraterone acetate plus prednisone therapy in patients with docetaxel-treated castration-resistant prostate cancer;Danila;J Clin Oncol,2010
5. Abiraterone and increased survival in metastatic prostate cancer;de Bono;N Engl J Med,2011
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2. PSA response to antiandrogen withdrawal: a systematic review and meta-analysis;Prostate Cancer and Prostatic Diseases;2021-02-18
3. Factores predictivos del síndrome de retirada de abiraterona;Actas Urológicas Españolas;2019-07
4. Predictive factors for abiraterone withdrawal syndrome;Actas Urológicas Españolas (English Edition);2019-07
5. Metastatic prostate cancer remains incurable, why?;Asian Journal of Urology;2019-01
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