IGF2BP3 is an essential N6-methyladenosine biotarget for suppressing ferroptosis in lung adenocarcinoma cells-Reference-Cited by-同舟云学术

IGF2BP3 is an essential N6-methyladenosine biotarget for suppressing ferroptosis in lung adenocarcinoma cells

Published:2022-12 Issue: Volume:17 Page:100503
ISSN:2590-0064
Container-title:Materials Today Bio
language:en
Short-container-title:Materials Today Bio

Author:

Xu Xin,Cui Jiangtao,Wang Hong,Ma Lifang,Zhang Xiao,Guo Wanxin,Xue Xiangfei,Wang Yikun,Qiu Shiyu,Tian Xiaoting,Miao Yayou,Wu Mengyi,Yu Yongchun,Xu Yunhua,Wang Jiayi^ORCID,Qiao Yongxia

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biomedical Engineering,Biomaterials,Bioengineering,Biotechnology

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1. COPZ1 regulates ferroptosis through NCOA4-mediated ferritinophagy in lung adenocarcinoma;Biochimica et Biophysica Acta (BBA) - General Subjects;2024-11

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4. IGF2BP3 suppresses ferroptosis in lung adenocarcinoma by m6A-dependent regulation of TFAP2A to transcriptionally activate SLC7A11/GPX4;Molecular and Cellular Biochemistry;2024-07-18

5. Systematic analysis of IGF2BP family members in non-small-cell lung cancer;Human Genomics;2024-06-12