Manganese exposure facilitates microglial JAK2-STAT3 signaling and consequent secretion of TNF-a and IL-1β to promote neuronal death-Reference-Cited by-同舟云学术

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Manganese exposure facilitates microglial JAK2-STAT3 signaling and consequent secretion of TNF-a and IL-1β to promote neuronal death

Published:2018-01 Issue: Volume:64 Page:195-203
ISSN:0161-813X
Container-title:NeuroToxicology
language:en
Short-container-title:NeuroToxicology

Author:

Yin Lifeng,Dai Qijun,Jiang Peipei,Zhu Lin,Dai Haifeng,Yao Zhigang,Liu Hua,Ma Xiaoping,Qu Lianxia,Jiang Junkang

Funder

National Natural Science Foundation of China

Key Laboratory of Public Health Safety

Ministry of Education

Nantong Jiangsu scientific research project

Publisher

Elsevier BV

Subject

Toxicology,General Neuroscience

Reference40 articles.

1. Inflammatory Process in Alzheimer's and Parkinson's Diseases: Central Role of Cytokines;Alam;Curr. Pharm. Des.,2016

2. Lipopolysaccharide-induced interleukin-6 production is controlled by glycogen synthase kinase-3 and STAT3 in the brain;Beurel;J. Neuroinflammation,2009

3. Manganese Neurotoxicity: behavioral disorders associated with dysfunctions in the basal ganglia and neurochemical transmission;Bouabid;J. Neurochem.,2015

4. Neurobehavioral functioning after cessation of manganese exposure: a follow-up after 14 years;Bouchard;Am. J. Ind. Med.,2007

5. Inhibition of nitric oxide production by the carbazole compound LCY-2-CHO via blockade of activator protein-1 and CCAAT/enhancer-binding protein activation in microglia;Chang;Biochem. Pharmacol.,2008

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