Downregulation of fatty acid oxidation led by Hilpda increases G2/M arrest/delay-induced kidney fibrosis-Reference-Cited by-同舟云学术

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Downregulation of fatty acid oxidation led by Hilpda increases G2/M arrest/delay-induced kidney fibrosis

Published:2023-06 Issue:5 Volume:1869 Page:166701
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Liu Limin,Liu Ting,Jia Rui,Zhang Lizi,Lv Zijian,He Zhixiong,Qu Yishan,Sun Shiren,Tai Fadao

Funder

National Natural Science Foundation of China

Shaanxi Province Natural Science Foundation

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference65 articles.

1. ATF6alpha downregulation of PPARalpha promotes lipotoxicity-induced tubulointerstitial fibrosis;Jao;Kidney Int.,2019

2. Autophagy activation contributes to lipid accumulation in tubular epithelial cells during kidney fibrosis;Yan;Cell death discovery,2018

3. The interdependence of renal epithelial and endothelial metabolism and cell state;Balzer;Sci. Signal.,2020

4. Gene and protein expressions and metabolomics exhibit activated redox signaling and wnt/beta-catenin pathway are associated with metabolite dysfunction in patients with chronic kidney disease;Chen;Redox Biol.,2017

5. Defective fatty acid oxidation in renal tubular epithelial cells has a key role in kidney fibrosis development;Kang;Nat. Med.,2015

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. C3aR Antagonist Alleviates C3a Induced Tubular Profibrotic Phenotype Transition via Restoring PPARα/CPT-1α Mediated Mitochondrial Fatty Acid Oxidation in Renin-Dependent Hypertension;Frontiers in Bioscience-Landmark;2023-10-17

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