Antigen presenting cell abnormalities in the Cln3 −/− mouse model of juvenile neuronal ceroid lipofuscinosis-Reference-Cited by-同舟云学术

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Antigen presenting cell abnormalities in the Cln3 −/− mouse model of juvenile neuronal ceroid lipofuscinosis

Published:2016-07 Issue:7 Volume:1862 Page:1324-1336
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Hersrud Samantha L.,Kovács Attila D.,Pearce David A.

Funder

National Institutes of Health (NIH)

Luke and Rachel Batten Foundation

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference97 articles.

1. Current state of clinical and morphological features in human NCL;Goebel;Brain Pathol.,2004

2. Late onset neurodegeneration in the Cln3−/− mouse model of juvenile neuronal ceroid lipofuscinosis is preceded by low level glial activation;Pontikis;Brain Res.,2004

3. Hippocampal pathology in the human neuronal ceroid-lipofuscinoses: distinct patterns of storage deposition, neurodegeneration and glial activation;Tyynela;Brain Pathol.,2004

4. Cerebellar defects in a mouse model of juvenile neuronal ceroid lipofuscinosis;Weimer;Brain Res.,2009

5. The juvenile Batten disease protein, CLN3, and its role in regulating anterograde and retrograde post-Golgi trafficking;Cotman;Clin. Lipidol.,2012

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2. Longitudinal Deep Multi-Omics Profiling in aCLN3^Δex7/8Minipig Model Reveals Novel Biomarker Signatures for Batten Disease;2023-09-22

3. A mouse mutant deficient in both neuronal ceroid lipofuscinosis‐associated proteins CLN3 and TPP1;Journal of Inherited Metabolic Disease;2023-04-26

4. Ocular Manifestations of Neuronal Ceroid Lipofuscinoses;Seminars in Ophthalmology;2021-06-09

5. The contribution of multicellular model organisms to neuronal ceroid lipofuscinosis research;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2020-09

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