Zebrafish as a Model for Human Ciliopathies-Reference-Cited by-同舟云学术

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Zebrafish as a Model for Human Ciliopathies

Published:2016-03 Issue:3 Volume:43 Page:107-120
ISSN:1673-8527
Container-title:Journal of Genetics and Genomics
language:en
Short-container-title:Journal of Genetics and Genomics

Author:

Song Zhu,Zhang Xiaoli,Jia Shuo,Yelick Pamela C.,Zhao Chengtian

Funder

National Natural Science Foundation of China

Shandong Provincial Natural Science Foundation

NIH/NIDCR

Publisher

Elsevier BV

Subject

Genetics,Molecular Biology

Reference137 articles.

1. IFT27, encoding a small GTPase component of IFT particles, is mutated in a consanguineous family with Bardet-Biedl syndrome;Aldahmesh;Hum. Mol. Genet.,2014

2. CRISPR/Cas9 and TALEN-mediated knock-in approaches in zebrafish;Auer;Methods,2014

3. Zebrafish ciliopathy screen plus human mutational analysis identifies C21orf59 and CCDC65 defects as causing primary ciliary dyskinesia;Austin-Tse;Am. J. Hum. Genet.,2013

4. KIAA0586 is mutated in Joubert syndrome;Bachmann-Gagescu;Hum. Mutat.,2015

5. The ciliopathy gene cc2d2a controls zebrafish photoreceptor outer segment development through a role in Rab8-dependent vesicle trafficking;Bachmann-Gagescu;Hum. Mol. Genet.,2011

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