ISPD Overexpression Enhances Ribitol-Induced Glycosylation of α-Dystroglycan in Dystrophic FKRP Mutant Mice-Reference-Cited by-同舟云学术

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ISPD Overexpression Enhances Ribitol-Induced Glycosylation of α-Dystroglycan in Dystrophic FKRP Mutant Mice

Published:2020-06 Issue: Volume:17 Page:271-280
ISSN:2329-0501
Container-title:Molecular Therapy - Methods & Clinical Development
language:en
Short-container-title:Molecular Therapy - Methods & Clinical Development

Author:

Cataldi Marcela P.,Blaeser Anthony,Lu Peijuan,Leroy Victoria,Lu Qi Long

Funder

Carolinas Muscular Dystrophy Research Endowment at the Atrium Health Foundation

Publisher

Elsevier BV

Subject

Genetics,Molecular Biology,Molecular Medicine

Reference28 articles.

1. The genetic and molecular basis of muscular dystrophy: roles of cell-matrix linkage in the pathogenesis;Kanagawa;J. Hum. Genet.,2006

2. Membrane organization of the dystrophin-glycoprotein complex;Ervasti;Cell,1991

3. A role for the dystrophin-glycoprotein complex as a transmembrane linker between laminin and actin;Ervasti;J. Cell Biol.,1993

4. Dystroglycan-alpha, a dystrophin-associated glycoprotein, is a functional agrin receptor;Gee;Cell,1994

5. Binding of the G domains of laminin alpha1 and alpha2 chains and perlecan to heparin, sulfatides, alpha-dystroglycan and several extracellular matrix proteins;Talts;EMBO J.,1999

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1. Dual FKRP/FST gene therapy normalizes ambulation, increases strength, decreases pathology, and amplifies gene expression in LGMDR9 mice;Molecular Therapy;2024-08

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3. Inhibitory CCK+ basket synapse defects in mouse models of dystroglycanopathy;eLife;2024-01-05

4. Inhibitory CCK+ basket synapse defects in mouse models of dystroglycanopathy;eLife;2024-01-05

5. Clinical and Genetic Characteristics of Limb-Girdle Muscular Dystrophy in Iranian Patients;Iranian Journal of Pediatrics;2023-11-12

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