The Origin of B Cell Recurrent Chromosomal Translocations: Proximity versus DNA Damage
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology
Reference9 articles.
1. Genome-wide Translocation Sequencing Reveals Mechanisms of Chromosome Breaks and Rearrangements in B Cells
2. AID-Targeting and Hypermutation of Non-Immunoglobulin Genes Does Not Correlate with Proximity to Immunoglobulin Genes in Germinal Center B Cells
3. DNA damage defines sites of recurrent chromosomal translocations in B lymphocytes
4. Translocation-Capture Sequencing Reveals the Extent and Nature of Chromosomal Rearrangements in B Lymphocytes
5. Comprehensive Mapping of Long-Range Interactions Reveals Folding Principles of the Human Genome
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1. Transcription factor binding at Ig enhancers is linked to somatic hypermutation targeting;European Journal of Immunology;2019-12-19
2. FGFR3 preferentially colocalizes with IGH in the interphase nucleus of multiple myeloma patient B-cells when FGFR3 is located outside of CT4;Genes, Chromosomes and Cancer;2016-08-11
3. Mechanisms of human lymphoid chromosomal translocations;Nature Reviews Cancer;2016-05-25
4. 3D view of chromosomes, DNA damage, and translocations;Current Opinion in Genetics & Development;2014-04
5. Molecular Pathways: Transcription Factories and Chromosomal Translocations;Clinical Cancer Research;2013-10-28
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