HMGB2 Loss upon Senescence Entry Disrupts Genomic Organization and Induces CTCF Clustering across Cell Types-Reference-Cited by-同舟云学术

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HMGB2 Loss upon Senescence Entry Disrupts Genomic Organization and Induces CTCF Clustering across Cell Types

Published:2018-05 Issue:4 Volume:70 Page:730-744.e6
ISSN:1097-2765
Container-title:Molecular Cell
language:en
Short-container-title:Molecular Cell

Author:

Zirkel Anne,Nikolic Milos,Sofiadis Konstantinos,Mallm Jan-Philipp,Brackley Chris A.,Gothe Henrike,Drechsel Oliver,Becker Christian,Altmüller Janine,Josipovic Natasa,Georgomanolis Theodore,Brant Lilija,Franzen Julia,Koker Mirjam,Gusmao Eduardo G.,Costa Ivan G.,Ullrich Roland T.,Wagner Wolfgang,Roukos Vassilis,Nürnberg Peter,Marenduzzo Davide,Rippe Karsten,Papantonis Argyris

Funder

IZKF

Else Kröner Fresenius Stiftung

DFG

CMMC

UoC Advanced Postdoc Grant

Fritz Thyssen Stiftung

DFG Major Research Instrumentation Program

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference67 articles.

1. Aberrant neural stem cell proliferation and increased adult neurogenesis in mice lacking chromatin protein HMGB2;Abraham;PLoS ONE,2013

2. A complex secretory program orchestrated by the inflammasome controls paracrine senescence;Acosta;Nat. Cell Biol.,2013

3. HMGB2 orchestrates the chromatin landscape of senescence-associated secretory phenotype gene loci;Aird;J. Cell Biol.,2016

4. Hi-C: a comprehensive technique to capture the conformation of genomes;Belton;Methods,2012

5. The DNA chaperone HMGB1 facilitates ACF/CHRAC-dependent nucleosome sliding;Bonaldi;EMBO J.,2002

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