NLRP3 Phosphorylation Is an Essential Priming Event for Inflammasome Activation-Reference-Cited by-同舟云学术

NLRP3 Phosphorylation Is an Essential Priming Event for Inflammasome Activation

Published:2017-10 Issue:1 Volume:68 Page:185-197.e6
ISSN:1097-2765
Container-title:Molecular Cell
language:en
Short-container-title:Molecular Cell

Author:

Song Nan,Liu Zhao-Shan,Xue Wen,Bai Zhao-Fang,Wang Qian-Yi,Dai Jiang,Liu Xin,Huang Yi-Jiao,Cai Hong,Zhan Xiao-Yan,Han Qiu-Ying,Wang Hongxia,Chen Yuan,Li Hui-Yan,Li Ai-Ling,Zhang Xue-Min,Zhou Tao,Li Tao

Funder

National Science and Technology Major Projects

China National Basic Research Program

National High-Tech R&D Program of China

China National Natural Science Foundation

Frontier Technology of Beijing S&T Commission

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference61 articles.

1. NALP3 forms an IL-1beta-processing inflammasome with increased activity in Muckle-Wells autoinflammatory disorder;Agostini;Immunity,2004

2. The NLRP3 inflammasome is released as a particulate danger signal that amplifies the inflammatory response;Baroja-Mazo;Nat. Immunol.,2014

3. iGLuc: A luciferase-based inflammasome and protease activity reporter;Bartok;Nat. Methods,2013

4. Cutting edge: NF-kappaB activating pattern recognition and cytokine receptors license NLRP3 inflammasome activation by regulating NLRP3 expression;Bauernfeind;J. Immunol.,2009

5. SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase;Bennett;Proc. Natl. Acad. Sci. USA,2001

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