Effects and mechanism of miR-23b on glucose-mediated epithelial-to-mesenchymal transition in diabetic nephropathy-Reference-Cited by-同舟云学术

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Effects and mechanism of miR-23b on glucose-mediated epithelial-to-mesenchymal transition in diabetic nephropathy

Published:2016-01 Issue: Volume:70 Page:149-160
ISSN:1357-2725
Container-title:The International Journal of Biochemistry & Cell Biology
language:en
Short-container-title:The International Journal of Biochemistry & Cell Biology

Author:

Liu Haifeng,Wang Xiaohua,Liu Shengfeng,Li Hongzhi,Yuan Xiaohuan,Feng Biao,Bai He,Zhao Binghai,Chu Yanhui,Li Hongjian

Funder

National Natural Sciences Foundation of China

Nature Science Foundation of Heilongjiang Province

Open Project Program of Heilongjiang Key Laboratory of Tissue Injury and Repair

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry

Reference45 articles.

1. Mapk and jak/stat pathways targeted by mir-23a and mir-23b in prostate cancer: computational and in vitro approaches;Aghaee-Bakhtiari;Tumour Biol.: J. Int. Soc. Oncodev. Biol. Med.,2015

2. Ellagic acid, an nf-kappab inhibitor, ameliorates renal function in experimental diabetic nephropathy;Ahad;Chem.-Biol. Interact.,2014

3. Phosphatidylinositol 3-kinase class ii alpha-isoform pi3k-c2alpha is required for transforming growth factor beta-induced smad signaling in endothelial cells;Aki;J. Biol. Chem.,2015

4. Lipoxins attenuate renal fibrosis by inducing let-7c and suppressing tgfbetar1;Brennan;J. Am. Soc. Nephrol.: JASN,2013

5. Mechanisms of endothelial to mesenchymal transition in the retina in diabetes;Cao;Investig. Ophthalmol. Vis. Sci.,2014

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