sFlt-1 and soluble endoglin concentrations in serum vs plasma in preterm preeclampsia: Are they interchangeable for biomarker studies?
Author:
Publisher
Elsevier BV
Subject
Obstetrics and Gynaecology,Internal Medicine
Reference16 articles.
1. Latest advances in understanding preeclampsia;Redman;Science,2005
2. Pre-eclampsia;Sibai;Lancet,2005
3. Excess placental soluble fms-like tyrosine kinase 1 (sFlt-1) may contribute to endothelial dysfunction, hypertension, and proteinuria in pre-eclampsia;Maynard;J. Clin. Invest.,2003
4. Soluble endoglin contributes to the pathogenesis of preeclampsia;Venkatesha;Nat Med.,2006
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1. An endogenous inhibitor of angiogenesis downregulated by hypoxia in human aortic valve stenosis promotes disease pathogenesis;Journal of Molecular and Cellular Cardiology;2023-01
2. Decidual mesenchymal stem/stromal cells from preeclamptic patients secrete endoglin, which at high levels inhibits endothelial cell attachment in vitro;Placenta;2022-08
3. Assessing the Role of Uric Acid as a Predictor of Preeclampsia;Frontiers in Physiology;2022-01-13
4. SOLUBLE ENDOGLIN AS AN EARLY PREDICTION MARKER OF INTRAUTERINE GROWTH RETARDATION;World of Medicine and Biology;2021
5. Decidual mesenchymal stem/stromal cell-derived extracellular vesicles ameliorate endothelial cell proliferation, inflammation, and oxidative stress in a cell culture model of preeclampsia;Pregnancy Hypertension;2020-10
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