Contractile impairment and structural alterations of skeletal muscles from knockout mice lacking type 1 and type 3 ryanodine receptors-Reference-Cited by-同舟云学术

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Contractile impairment and structural alterations of skeletal muscles from knockout mice lacking type 1 and type 3 ryanodine receptors

Published:1998-01-30 Issue:2 Volume:422 Page:160-164
ISSN:0014-5793
Container-title:FEBS Letters
language:en
Short-container-title:

Author:

Barone Virginia,Bertocchini Federica,Bottinelli Roberto,Protasi Feliciano,Allen Paul D.,Franzini Armstrong Clara,Reggiani Carlo,Sorrentino Vincenzo

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1016/S0014-5793(98)00003-9/fullpdf

Reference19 articles.

1. Excitation-contraction uncoupling and muscular degeneration in mice lacking functional skeletal muscle ryanodine-receptor gene

2. Enhanced dihydropyridine receptor channel activity in the presence of ryanodine receptor

3. Electrical properties of normal and dysgenib mouse skeletal muscle in culture

4. Evidence for dysfunction in the regulation of cytosolic Ca2+ in excitation-contraction uncoupled dysgenic muscle

5. Expression of a Ryanodine Receptor-Ca 2+ Channel that is Regulated by TGF-β

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1. Preclinical model systems of ryanodine receptor 1-related myopathies and malignant hyperthermia: a comprehensive scoping review of works published 1990–2019;Orphanet Journal of Rare Diseases;2020-05-07

2. Blocking skeletal muscle DHPRs/Ryr1 prevents neuromuscular synapse loss in mutant mice deficient in type III Neuregulin 1 (CRD-Nrg1);PLOS Genetics;2019-03-14

3. Modelos animais ajudando a decifrar doenças neuromusculares humanas;Revista Neurociências;2019-01-23

4. STAC3 incorporation into skeletal muscle triads occurs independent of the dihydropyridine receptor;Journal of Cellular Physiology;2018-08-02

5. Calcium Influx and Release Cooperatively Regulate AChR Patterning and Motor Axon Outgrowth during Neuromuscular Junction Formation;Cell Reports;2018-06

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