STAC3 incorporation into skeletal muscle triads occurs independent of the dihydropyridine receptor
Author:
Affiliation:
1. Department of Physiology Medical University, Innsbruck Innsbruck Austria
Funder
Austrian Science Fund
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.26767
Reference26 articles.
1. Bridging the myoplasmic gap II: more recent advances in skeletal muscle excitation–contraction coupling
2. Contractile impairment and structural alterations of skeletal muscles from knockout mice lacking type 1 and type 3 ryanodine receptors
3. Structural evidence for direct interaction between the molecular components of the transverse tubule/sarcoplasmic reticulum junction in skeletal muscle.
4. STAC proteins associate to the IQ domain of CaV1.2 and inhibit calcium-dependent inactivation
5. STAC3 stably interacts through its C1 domain with CaV1.1 in skeletal muscle triads
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