Endothelial Nitric Oxide Synthase-Induced Hypertrophy and Vascular Dysfunction Contribute to the Left Ventricular Dysfunction in Caveolin-1−/− Mice-Reference-Cited by-同舟云学术

Endothelial Nitric Oxide Synthase-Induced Hypertrophy and Vascular Dysfunction Contribute to the Left Ventricular Dysfunction in Caveolin-1−/− Mice

Published:2017-12 Issue:12 Volume:33 Page:1716-1724
ISSN:0828-282X
Container-title:Canadian Journal of Cardiology
language:en
Short-container-title:Canadian Journal of Cardiology

Author:

Ebner Annette,Kuerbis Nadine,Brandt Aljoscha,Zatschler Birgit,Weinert Sönke,Poitz David M.,Ebner Bernd,Augstein Antje,Wunderlich Carsten,El-Armouche Ali,Strasser Ruth H.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference26 articles.

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2. Caveolin-1 and caveolin-3 form heterooligomeric complexes in atrial cardiac myocytes that are required for doxorubicin-induced apoptosis;Volonte;Am J Physiol Heart Circ Physiol,2008

3. Caveolin-1 exists and may function in cardiomyocytes;Cho;Can J Physiol Pharmacol,2010

4. Dexrazoxane prevents the development of the impaired cardiac phenotype in caveolin-1-disrupted mice;Polanski;J Cardiovasc Pharmacol,2013

5. Disruption of caveolin-1 leads to enhanced nitrosative stress and severe systolic and diastolic heart failure;Wunderlich;Biochem Biophys Res Commun,2006

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3. Overexpression of endothelial β 3 ‐adrenergic receptor induces diastolic dysfunction in rats;ESC Heart Failure;2020-10-09

4. Caveolin as a Novel Potential Therapeutic Target in Cardiac and Vascular Diseases: A Mini Review;Aging and disease;2020

5. NOS3 Deletion in Cav1 Deficient Mice Decreases Drug Sensitivity to a Nitric Oxide Donor and Two Nitric Oxide Synthase Inhibitors;Investigative Opthalmology & Visual Science;2019-09-23