Febrile Temperature Critically Controls the Differentiation and Pathogenicity of T Helper 17 Cells-Reference-Cited by-同舟云学术

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Febrile Temperature Critically Controls the Differentiation and Pathogenicity of T Helper 17 Cells

Published:2020-02 Issue:2 Volume:52 Page:328-341.e5
ISSN:1074-7613
Container-title:Immunity
language:en
Short-container-title:Immunity

Author:

Wang Xiaohu,Ni Lu,Wan Siyuan,Zhao Xiaohong,Ding Xiao,Dejean Anne,Dong Chen

Funder

National Key Research and Development Program

National Natural Science Foundation

Beijing Municipal Commission of Science and Technology

Beijing Natural Science Foundation

Publisher

Elsevier BV

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Reference56 articles.

1. The heat-stable antigen determines pathogenicity of self-reactive T cells in experimental autoimmune encephalomyelitis;Bai;J. Clin. Invest.,2000

2. Loss of T-bet, but not STAT1, prevents the development of experimental autoimmune encephalomyelitis;Bettelli;J. Exp. Med.,2004

3. Effect of targeted disruption of STAT4 and STAT6 on the induction of experimental autoimmune encephalomyelitis;Chitnis;J. Clin. Invest.,2001

4. Differential requirements for Smad4 in TGFbeta-dependent patterning of the early mouse embryo;Chu;Development,2004

5. Sumoylation by Ubc9 regulates the stem cell compartment and structure and function of the intestinal epithelium in mice;Demarque;Gastroenterology,2011

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