Dysbiosis exacerbates colitis by promoting ubiquitination and accumulation of the innate immune adaptor STING in myeloid cells-Reference-Cited by-同舟云学术

Dysbiosis exacerbates colitis by promoting ubiquitination and accumulation of the innate immune adaptor STING in myeloid cells

Published:2021-06 Issue:6 Volume:54 Page:1137-1153.e8
ISSN:1074-7613
Container-title:Immunity
language:en
Short-container-title:Immunity

Author:

Shmuel-Galia Liraz,Humphries Fiachra,Lei Xuqiu,Ceglia Simona,Wilson Ruth,Jiang Zhaozhao,Ketelut-Carneiro Natalia,Foley Sage E.,Pechhold Susanne,Houghton JeanMarie,Muneeruddin Khaja,Shaffer Scott A.,McCormick Beth A.,Reboldi Andrea,Ward Doyle,Marshak-Rothstein Ann,Fitzgerald Katherine A.

Funder

Crohn's and Colitis Foundation of America

National Institutes of Health

Publisher

Elsevier BV

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Reference66 articles.

1. Cytosolic-DNA-mediated, STING-dependent proinflammatory gene induction necessitates canonical NF-κB activation through TBK1;Abe;J. Virol.,2014

2. ATG16L1 orchestrates interleukin-22 signaling in the intestinal epithelium via cGAS-STING;Aden;J. Exp. Med.,2018

3. STING-Dependent Signaling Underlies IL-10 Controlled Inflammatory Colitis;Ahn;Cell Rep.,2017

4. Microbial imbalance in inflammatory bowel disease patients at different taxonomic levels;Alam;Gut Pathog.,2020

5. A Human Gain-of-Function STING Mutation Causes Immunodeficiency and Gammaherpesvirus-Induced Pulmonary Fibrosis in Mice;Bennion;J. Virol.,2019

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