Targeting STING oligomerization with licochalcone D ameliorates STING-driven inflammatory diseases
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Publisher
Springer Science and Business Media LLC
Link
https://link.springer.com/content/pdf/10.1007/s11427-024-2703-6.pdf
Reference48 articles.
1. Abe, T., and Barber, G.N. (2014). Cytosolic-DNA-mediated, STING-dependent proinflammatory gene induction necessitates canonical NF-κB activation through TBK1. J Virol 88, 5328–5341.
2. Ablasser, A., Goldeck, M., Cavlar, T., Deimling, T., Witte, G., Röhl, I., Hopfner, K.P., Ludwig, J., and Hornung, V. (2013). cGAS produces a 2′-5′-linked cyclic dinucleotide second messenger that activates STING. Nature 498, 380–384.
3. Ahn, J., Ruiz, P., and Barber, G.N. (2014). Intrinsic self-DNA triggers inflammatory disease dependent on STING. J Immunol 193, 4634–4642.
4. Ahn, J., Son, S., Oliveira, S.C., and Barber, G.N. (2017). STING-dependent signaling underlies IL-10 controlled inflammatory colitis. Cell Rep 21, 3873–3884.
5. Barber, G.N. (2015). STING: infection, inflammation and cancer. Nat Rev Immunol 15, 760–770.
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