Data on COA-Cl administration to the APP/PS2 double-transgenic mouse model of Alzheimer׳s disease: Improved hippocampus-dependent learning and unchanged spontaneous physical activity-Reference-Cited by-同舟云学术

Data on COA-Cl administration to the APP/PS2 double-transgenic mouse model of Alzheimer׳s disease: Improved hippocampus-dependent learning and unchanged spontaneous physical activity

Published:2018-10 Issue: Volume:20 Page:1877-1883
ISSN:2352-3409
Container-title:Data in Brief
language:en
Short-container-title:Data in Brief

Author:

Kishimoto Yasushi^ORCID,Tsukamoto Ikuko,Nishigawa Atsuko,Nishimoto Akiko,Kirino Yutaka,Kato Yoshihisa,Konishi Ryoji,Maruyama Tokumi,Sakakibara Norikazu

Funder

JSPS KAKENHI

Publisher

Elsevier BV

Subject

Multidisciplinary

Reference8 articles.

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2. Involvement of S1P1 receptor pathway in angiogenic effects of a novel adenosine-like nucleic acid analog COA-Cl in cultured human vascular endothelial cells;Igarashi;Pharmacol. Res. Perspect.,2014

3. Presenilin 2 mutation accelerates the onset of impairment in trace eyeblink conditioning in a mouse model of Alzheimer׳s disease overexpressing human mutant amyloid precursor protein;Kishimoto;Neurosci. Lett.,2013

4. Establishment of an Alzheimer׳s disease model with latent herpesvirus infection using PS2 and Tg2576 double transgenic mice;Tanaka;Exp. Anim.,2018

5. Presenilin-2 mutation causes early amyloid accumulation and memory impairment in a transgenic mouse model of Alzheimer׳s disease;Toda;J. Biomed. Biotechnol.,2011

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1. Raman Spectroscopic and DFT Study of COA-Cl and Its Analogues;The Journal of Physical Chemistry A;2023-02-21

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