Establishment of an Alzheimer’s disease model with latent herpesvirus infection using PS2 and Tg2576 double transgenic mice
Author:
Affiliation:
1. Center for Experimental Animals, Fukuoka University, 7-45-1 Nanakuma, Jonan-ku, Fukuoka-shi, Fukuoka 814-0180, Japan
2. Animal-care Co., Ltd., 5-18-14 Shinjuku, Shinjuku-ku, Tokyo 160-0022, Japan
Publisher
Japanese Association for Laboratory Animal Science
Subject
General Veterinary,General Biochemistry, Genetics and Molecular Biology,Animal Science and Zoology,General Medicine
Link
https://www.jstage.jst.go.jp/article/expanim/67/2/67_17-0066/_pdf
Reference53 articles.
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2. 2. Amano N., Matsuishi T., Suzuki K., Yokoi S., Saitoh A., and Yagishita S. 1984. [Ultrastructural study of the neuronal lipofuscin--an autopsy case of familial Alzheimer’s disease]. No To Shinkei 36: 657–663. (in Japanese)
3. 3. Arnáiz E. and Almkvist O. 2003. Neuropsychological features of mild cognitive impairment and preclinical Alzheimer’s disease. Acta Neurol. Scand. Suppl. 107: 34–41.
4. 4. Bäckman L., Jones S., Berger A.K., Laukka E.J., and Small B.J. 2004. Multiple cognitive deficits during the transition to Alzheimer’s disease. J. Intern. Med. 256: 195–204.
5. 5. Ball M.J. 1982. Limbic predilection in Alzheimer dementia: is reactivated herpesvirus involved? Can. J. Neurol. Sci. 9: 303–306.
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