Lrp5 and Lrp6 redundantly control skeletal development in the mouse embryo-Reference-Cited by-同舟云学术

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Lrp5 and Lrp6 redundantly control skeletal development in the mouse embryo

Published:2011-11 Issue:2 Volume:359 Page:222-229
ISSN:0012-1606
Container-title:Developmental Biology
language:en
Short-container-title:Developmental Biology

Author:

Joeng Kyu Sang,Schumacher Cassie A.,Zylstra-Diegel Cassandra R.,Long Fanxin,Williams Bart O.

Funder

NIH

Van Andel Research Institute

Publisher

Elsevier BV

Subject

Cell Biology,Developmental Biology,Molecular Biology

Reference39 articles.

1. Increased bone density in sclerosteosis is due to the deficiency of a novel secreted protein (SOST);Balemans;Hum. Mol. Genet.,2001

2. Identification of a 52kb deletion downstream of the SOST gene in patients with van Buchem disease;Balemans;J. Med. Genet.,2002

3. Regulation of osteoblastogenesis and bone mass by Wnt10b;Bennett;Proc. Natl. Acad. Sci. U. S. A.,2005

4. High bone density due to a mutation in LDL-receptor-related protein 5;Boyden;N. Engl. J. Med.,2002

5. Inactivation of the beta-catenin gene by Wnt1-Cre-mediated deletion results in dramatic brain malformation and failure of craniofacial development;Brault;Development,2001

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