Fetal and postnatal lung defects reveal a novel and required role for Fgf8 in lung development-Reference-Cited by-同舟云学术

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Fetal and postnatal lung defects reveal a novel and required role for Fgf8 in lung development

Published:2010-11 Issue:1 Volume:347 Page:92-108
ISSN:0012-1606
Container-title:Developmental Biology
language:en
Short-container-title:Developmental Biology

Author:

Yu Shibin,Poe Bryan,Schwarz Margaret,Elliot Sarah A.,Albertine Kurt H.,Fenton Stephen,Garg Vidu,Moon Anne M.

Funder

American Lung Association Career Investigator Award

NIH

Publisher

Elsevier BV

Subject

Cell Biology,Developmental Biology,Molecular Biology

Reference98 articles.

1. Conditional gene inactivation reveals roles for Fgf10 and Fgfr2 in establishing a normal pattern of epithelial branching in the mouse lung;Abler;Dev. Dyn.,2009

2. Fgf8 is required for pharyngeal arch and cardiovascular development in the mouse;Abu-Issa;Development,2002

3. A morphologic and morphometric analysis of fetal lung development in the sheep;Alcorn;Anat. Rec.,1981

4. Role of ETS transcription factors in the hypoxia-inducible factor-2 target gene selection;Aprelikova;Cancer Res.,2006

5. Fgfr2 is required for limb outgrowth and lung-branching morphogenesis;Arman;Proc. Natl Acad. Sci. USA,1999

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