Sf3b4-depleted Xenopus embryos: A model to study the pathogenesis of craniofacial defects in Nager syndrome-Reference-Cited by-同舟云学术

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Sf3b4-depleted Xenopus embryos: A model to study the pathogenesis of craniofacial defects in Nager syndrome

Published:2016-07 Issue:2 Volume:415 Page:371-382
ISSN:0012-1606
Container-title:Developmental Biology
language:en
Short-container-title:Developmental Biology

Author:

Devotta Arun,Juraver-Geslin Hugo,Gonzalez Jose Antonio,Hong Chang-Soo,Saint-Jeannet Jean-Pierre

Funder

National Institutes of Health to J-P S-J

Publisher

Elsevier BV

Subject

Cell Biology,Developmental Biology,Molecular Biology

Reference60 articles.

1. Nager acrofacial dysostosis: male-to-male transmission in 2 families;Aylsworth;Am. J. Med. Genet.,1991

2. The zebrafish sf3b1b460 mutant reveals differential requirements for sf3b1 pre-mRNA processing gene during neural crest development;An;Int. J. Dev. Biol.,2012

3. Sox10 regulates the development of neural crest-derived melanocytes in Xenopus;Aoki;Dev. Biol.,2003

4. Induction and Specification of Neural Crest Cells: Extracellular Signals and Transcriptional Switches;Bae,2014

5. Hoxa2 knockdown in Xenopus results in hyoid to mandibular homeosis;Baltzinger;Dev. Dyn.,2005

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