Understanding the physiological role of NaV1.9: Challenges and opportunities for pain modulation
Author:
Publisher
Elsevier BV
Subject
Pharmacology (medical),Pharmacology
Reference140 articles.
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3. Membrane cholesterol depletion as a trigger of Nav1.9 channel-mediated inflammatory pain;Amsalem;EMBO Journal,2018
4. Voltage gated sodium channels as drug discovery targets;Bagal;Channels (Austin, Tex.),2015
5. Protein kinase C-alpha upregulates sodium channel Nav1.9 in nociceptive dorsal root ganglion neurons in an inflammatory arthritis pain model of rat;Bai;Journal of Cellular Biochemistry,2020
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1. Genetic Analysis of SCN11A, SCN10A, and SCN9A in Familial Episodic Pain Syndrome (FEPS) in Japan and Proposal of Clinical Diagnostic Criteria;International Journal of Molecular Sciences;2024-06-21
2. Anxiety and dysautonomia symptoms in patients with a NaV1.7 mutation and the potential benefits of low-dose short-acting guanfacine;Clinical Autonomic Research;2023-12-08
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