Membrane cholesterol depletion as a trigger of Nav1.9 channel‐mediated inflammatory pain-Reference-Cited by-同舟云学术

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Membrane cholesterol depletion as a trigger of Nav1.9 channel‐mediated inflammatory pain

Published:2018-02-19 Issue:8 Volume:37 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Amsalem Muriel¹,Poilbout Corinne¹,Ferracci Géraldine¹,Delmas Patrick¹^ORCID,Padilla Francoise¹

Affiliation:

1. CNRS Laboratoire de Neuroscience Cognitive (LNC) UMR 7291 Aix‐Marseille‐Université Marseille Cedex 3 France

Funder

Centre National de la Recherche Scientifique

Agence Nationale de la Recherche

Fondation pour la Recherche Médicale

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201797349

Reference68 articles.

1. The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways

2. Role of Protein Kinase A in the Maintenance of Inflammatory Pain

3. Chronic Hypersensitivity For Inflammatory Nociceptor Sensitization Mediated by the ε Isozyme of Protein Kinase C

4. The Voltage-Gated Sodium Channel Nav1.9 Is an Effector of Peripheral Inflammatory Pain Hypersensitivity

5. Bilayer Thickness and Membrane Protein Function: An Energetic Perspective

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