Involvement of JAK/STAT signaling in the effect of cornel iridoid glycoside on experimental autoimmune encephalomyelitis amelioration in rats-Reference-Cited by-同舟云学术

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Involvement of JAK/STAT signaling in the effect of cornel iridoid glycoside on experimental autoimmune encephalomyelitis amelioration in rats

Published:2014-09 Issue:1-2 Volume:274 Page:28-37
ISSN:0165-5728
Container-title:Journal of Neuroimmunology
language:en
Short-container-title:Journal of Neuroimmunology

Author:

Yin Linlin,Chen Yongyan,Qu Zhao,Zhang Li,Wang Qi,Zhang Qi,Li Lin

Funder

National Natural Science Foundation of China

Beijing Nova Star Program in Science and Technology

Capital Medical Development Special Program

Beijing Organization Department of Municipal Party Committee Project for Highly Talented Man

Publisher

Elsevier BV

Subject

Clinical Neurology,Neurology,Immunology,Immunology and Allergy

Reference54 articles.

1. Neurogenesis and neuroprotection induced by peripheral immunomodulatory treatment of experimental autoimmune encephalomyelitis;Aharoni;J. Neurosci.,2005

2. The role of STATs in apoptosis;Battle;Curr. Mol. Med.,2002

3. NF-kappaB family of transcription factors: central regulators of innate and adaptive immune functions;Caamaño;Clin. Microbiol. Rev.,2002

4. Clinical and histological findings in proteolipid protein-induced experimental autoimmune encephalomyelitis (EAE) in the Lewis rat. Distribution of demyelination differs from that in EAE induced by other antigens;Chalk;J. Neurol. Sci.,1994

5. Regulatory properties of copolymer I in Th17 differentiation by altering STAT3 phosphorylation;Chen;J. Immunol.,2009

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