Heterozygous Spink1 Deficiency Promotes Trypsin-dependent Chronic Pancreatitis in Mice-Reference-Cited by-同舟云学术

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Heterozygous Spink1 Deficiency Promotes Trypsin-dependent Chronic Pancreatitis in Mice

Published:2024 Issue:3 Volume:18 Page:101361
ISSN:2352-345X
Container-title:Cellular and Molecular Gastroenterology and Hepatology
language:en
Short-container-title:Cellular and Molecular Gastroenterology and Hepatology

Author:

Demcsák Alexandra,Sahin-Tóth Miklós^ORCID

Funder

National Pancreas Foundation

National Institutes of Health

U.S. Department of Defense

NIDDK Division of Diabetes Endocrinology and Metabolic Diseases

Hirshberg Foundation for Pancreatic Cancer Research

Publisher

Elsevier BV

Reference37 articles.

1. Activation of pancreatic zymogens. Normal activation, premature intrapancreatic activation, protective mechanisms against inappropriate activation;Rinderknecht;Dig Dis Sci,1986

2. Genetic risk in chronic pancreatitis: the trypsin-dependent pathway;Hegyi;Dig Dis Sci,2017

3. Defective binding of SPINK1 variants is an uncommon mechanism for impaired trypsin inhibition in chronic pancreatitis;Szabó;J Biol Chem,2021

4. Inhibition of mouse trypsin isoforms by SPINK1 and effect of human pancreatitis-associated mutations;Morales Granda;Pancreatology,2023

5. Mutations in the gene encoding the serine protease inhibitor, Kazal type 1 are associated with chronic pancreatitis;Witt;Nat Genet,2000

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Secretagogue-induced pancreatitis in mice devoid of chymotrypsin;American Journal of Physiology-Gastrointestinal and Liver Physiology;2024-09-01

2. Alu insertion-mediated dsRNA structure formation with pre-existing Alu elements as a disease-causing mechanism;The American Journal of Human Genetics;2024-09

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