Polydatin prevents fructose-induced liver inflammation and lipid deposition through increasing miR-200a to regulate Keap1/Nrf2 pathway-Reference-Cited by-同舟云学术

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Polydatin prevents fructose-induced liver inflammation and lipid deposition through increasing miR-200a to regulate Keap1/Nrf2 pathway

Published:2018-09 Issue: Volume:18 Page:124-137
ISSN:2213-2317
Container-title:Redox Biology
language:en
Short-container-title:Redox Biology

Author:

Zhao Xiao-Juan,Yu Han-Wen,Yang Yan-Zi,Wu Wen-Yuan,Chen Tian-Yu,Jia Ke-Ke,Kang Lin-Lin,Jiao Rui-Qing,Kong Ling-Dong

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Organic Chemistry,Biochemistry,Clinical Biochemistry

Reference36 articles.

1. Dietary fructose in nonalcoholic fatty liver disease;Vos;Hepatology,2013

2. Reactive oxygen species-induced TXNIP drives fructose-mediated hepatic inflammation and lipid accumulation through NLRP3 inflammasome activation;Zhang;Antioxid. Redox Signal.,2015

3. Oxidative stress as a critical factor in nonalcoholic fatty liver disease pathogenesis;Spahis;Antioxid. Redox Signal.,2017

4. The Keap1-Nrf2 stress response pathway promotes mitochondrial hyperfusion through degradation of the mitochondrial fission protein Drp1;Sabouny;Antioxid. Redox Signal.,2017

5. Resveratrol attenuates testicular apoptosis in type 1 diabetic mice: role of Akt-mediated Nrf2 activation and p62-dependent Keap1 degradation;Zhao;Redox Biol.,2018

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