Knockout of dihydrofolate reductase in mice induces hypertension and abdominal aortic aneurysm via mitochondrial dysfunction-Reference-Cited by-同舟云学术

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Knockout of dihydrofolate reductase in mice induces hypertension and abdominal aortic aneurysm via mitochondrial dysfunction

Published:2019-06 Issue: Volume:24 Page:101185
ISSN:2213-2317
Container-title:Redox Biology
language:en
Short-container-title:Redox Biology

Author:

Li Qiang,Youn Ji Youn,Siu Kin Lung,Murugesan Priya,Zhang Yixuan,Cai Hua

Funder

National Institutes of Health

American Heart Association

Publisher

Elsevier BV

Subject

Organic Chemistry,Biochemistry,Clinical Biochemistry

Reference54 articles.

1. Identification and characterization of an inborn error of metabolism caused by dihydrofolate reductase deficiency;Banka;Am. J. Hum. Genet.,2011

2. Endothelial dihydrofolate reductase: critical for nitric oxide bioavailability and role in angiotensin II uncoupling of endothelial nitric oxide synthase;Chalupsky;Proc. Natl. Acad. Sci. U. S. A,2005

3. Critical role for tetrahydrobiopterin recycling by dihydrofolate reductase in regulation of endothelial nitric-oxide synthase coupling: relative importance of the de novo biopterin synthesis versus salvage pathways;Crabtree;J. Biol. Chem.,2009

4. Dihydrofolate reductase protects endothelial nitric oxide synthase from uncoupling in tetrahydrobiopterin deficiency;Crabtree;Free Radic. Biol. Med.,2011

5. Attenuation of angiotensin II signaling recouples eNOS and inhibits nonendothelial NOX activity in diabetic mice;Oak;Diabetes,2007

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