Inhibition of glycogen synthase kinase-3 enhances NRF2 protein stability, nuclear localisation and target gene transcription in pancreatic beta cells-Reference-Cited by-同舟云学术

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Inhibition of glycogen synthase kinase-3 enhances NRF2 protein stability, nuclear localisation and target gene transcription in pancreatic beta cells

Published:2024-05 Issue: Volume:71 Page:103117
ISSN:2213-2317
Container-title:Redox Biology
language:en
Short-container-title:Redox Biology

Author:

Patibandla Chinmai^ORCID,van Aalten Lidy,Dinkova-Kostova Albena T.,Honda Tadashi,Cuadrado Antonio,Fernández-Ginés Raquel,McNeilly Alison D.,Hayes John D.,Cantley James,Sutherland Calum

Funder

Diabetes UK

MICINN

Publisher

Elsevier BV

Reference58 articles.

1. The Lancet, Diabetes: a defining disease of the 21st century;Lancet,2023

2. The role of oxidative stress in pancreatic β cell dysfunction in diabetes;Eguchi;Int. J. Mol. Sci.,2021

3. The beta cell in type 2 diabetes;Christensen;Curr. Diabetes Rep.,2019

4. Oxidative stress leads to β-cell dysfunction through loss of β-cell identity;Leenders;Front. Immunol.,2021

5. Therapeutic targeting of the NRF2 and KEAP1 partnership in chronic diseases;Cuadrado;Nat. Rev. Drug Discov.,2019

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Potential role of molecular hydrogen therapy on oxidative stress and redox signaling in chronic kidney disease;Biomedicine & Pharmacotherapy;2024-07

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