The tuberous sclerosis complex model Eker (TSC2+/−) rat exhibits hyperglycemia and hyperketonemia due to decreased glycolysis in the liver-Reference-Cited by-同舟云学术

The tuberous sclerosis complex model Eker (TSC2+/−) rat exhibits hyperglycemia and hyperketonemia due to decreased glycolysis in the liver

Published:2016-01 Issue: Volume:590 Page:48-55
ISSN:0003-9861
Container-title:Archives of Biochemistry and Biophysics
language:en
Short-container-title:Archives of Biochemistry and Biophysics

Author:

Aizawa Yumi,Shirai Tomomi,Kobayashi Toshiyuki,Hino Okio,Tsujii Yoshimasa,Inoue Hirofumi,Kazami Machiko,Tadokoro Tadahiro,Suzuki Tsukasa,Kobayashi Ken-Ichi,Yamamoto Yuji

Funder

Tokyo University of Agriculture

Publisher

Elsevier BV

Subject

Molecular Biology,Biochemistry,Biophysics

Reference36 articles.

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3. Signal integration by mTORC1 coordinates nutrient input with biosynthetic output;Dibble;Nat. Cell Biol.,2013

4. A germline insertion in the tuberous sclerosis (Tsc2) gene gives rise to the Eker rat model of dominantly inherited cancer;Kobayashi;Nat. Genet.,1995

5. Distribution of Tsc2 protein in various normal rat tissues and renal tumours of Tsc2 mutant (Eker) rat detected by immunohistochemistry;Fukuda;Virchows Arch.,1999

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