In Vitro Models of GJB2-Related Hearing Loss Recapitulate Ca2+ Transients via a Gap Junction Characteristic of Developing Cochlea-Reference-Cited by-同舟云学术

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In Vitro Models of GJB2-Related Hearing Loss Recapitulate Ca2+ Transients via a Gap Junction Characteristic of Developing Cochlea

Published:2016-12 Issue:6 Volume:7 Page:1023-1036
ISSN:2213-6711
Container-title:Stem Cell Reports
language:en
Short-container-title:Stem Cell Reports

Author:

Fukunaga Ichiro,Fujimoto Ayumi,Hatakeyama Kaori,Aoki Toru,Nishikawa Atena,Noda Tetsuo,Minowa Osamu,Kurebayashi Nagomi,Ikeda Katsuhisa,Kamiya Kazusaku

Funder

JSPS

MEXT

AMED

Promotion and mutual aid corporation

Terumo Life Science Foundation

Takeda Science Foundation

Publisher

Elsevier BV

Subject

Cell Biology,Developmental Biology,Genetics,Biochemistry

Reference38 articles.

1. Connexins 26 and 30 are co-assembled to form gap junctions in the cochlea of mice;Ahmad;Biochem. Biophys. Res. Commun.,2003

2. ATP release through connexin hemichannels and gap junction transfer of second messengers propagate Ca2+ signals across the inner ear;Anselmi;Proc. Natl. Acad. Sci. USA,2008

3. Autosomal dominant prelingual hearing loss with palmoplantar keratoderma syndrome: variability in clinical expression from mutations of R75W and R75Q in the GJB2 gene;Birkenhager;Am. J. Med. Genet. A.,2010

4. Pharmacological properties of homomeric and heteromeric pannexin hemichannels expressed in Xenopus oocytes;Bruzzone;J. Neurochem.,2005

5. Connexin-26-associated deafness: phenotypic variability and progression of hearing loss;Chan;Genet. Med.,2010

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