Macrophage Polarization and Insulin Resistance: PPARγ in Control
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Physiology
Reference23 articles.
1. IKK-β links inflammation to obesity-induced insulin resistance
2. Conditional Knockout of Macrophage PPARγIncreases Atherosclerosis in C57BL/6 and Low-Density Lipoprotein Receptor–Deficient Mice
3. Oxidized Lipid-Driven Chemokine Receptor Switch, CCR2 to CX3CR1, Mediates Adhesion of Human Macrophages to Coronary Artery Smooth Muscle Cells Through a Peroxisome Proliferator-Activated Receptor γ–Dependent Pathway
4. Decreased lesion formation in CCR2−/− mice reveals a role for chemokines in the initiation of atherosclerosis
5. PPARγ Activation Primes Human Monocytes into Alternative M2 Macrophages with Anti-inflammatory Properties
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3. Tumor necrosis factor receptor-associated factor 5 protects against intimal hyperplasia by regulation of macrophage polarization via directly targeting PPARγ;Inflammation Research;2024-04-20
4. Immune dysregulation and macrophage polarization in peri-implantitis;Frontiers in Bioengineering and Biotechnology;2024-01-29
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