High Glucose Triggers Nucleotide Imbalance through O-GlcNAcylation of Key Enzymes and Induces KRAS Mutation in Pancreatic Cells-Reference-Cited by-同舟云学术

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High Glucose Triggers Nucleotide Imbalance through O-GlcNAcylation of Key Enzymes and Induces KRAS Mutation in Pancreatic Cells

Published:2019-06 Issue:6 Volume:29 Page:1334-1349.e10
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

Hu Chun-Mei,Tien Sui-Chih,Hsieh Ping-Kun,Jeng Yung-Ming,Chang Ming-Chu,Chang Yu-Ting,Chen Yi-Ju,Chen Yu-Ju,Lee Eva Y.-H.P.,Lee Wen-Hwa

Funder

Academia Sinica

Ministry of Science and Technology

Ministry of Education

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference60 articles.

1. Prevalence of diabetes mellitus in pancreatic cancer compared to common cancers;Aggarwal;Pancreas,2013

2. Activated Kras and Ink4a/Arf deficiency cooperate to produce metastatic pancreatic ductal adenocarcinoma;Aguirre;Genes Dev.,2003

3. Brain energy metabolism: focus on astrocyte-neuron metabolic cooperation;Bélanger;Cell Metab.,2011

4. Pancreatic cancer genomes reveal aberrations in axon guidance pathway genes;Biankin;Nature,2012

5. A little sugar goes a long way: the cell biology of O-GlcNAc;Bond;J. Cell Biol.,2015

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