Molecular and Genetic Crosstalks between mTOR and ERRα Are Key Determinants of Rapamycin-Induced Nonalcoholic Fatty Liver-Reference-Cited by-同舟云学术

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Molecular and Genetic Crosstalks between mTOR and ERRα Are Key Determinants of Rapamycin-Induced Nonalcoholic Fatty Liver

Published:2013-04 Issue:4 Volume:17 Page:586-598
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

Chaveroux Cédric,Eichner Lillian J.,Dufour Catherine R.,Shatnawi Aymen,Khoutorsky Arkady,Bourque Guillaume,Sonenberg Nahum,Giguère Vincent

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference46 articles.

1. A nuclear transport signal in mammalian target of rapamycin is critical for its cytoplasmic signaling to S6 kinase 1;Bachmann;J. Biol. Chem.,2006

2. Major involvement of mTOR in the PPARγ-induced stimulation of adipose tissue lipid uptake and fat accretion;Blanchard;J. Lipid Res.,2012

3. Yin Yang 1 deficiency in skeletal muscle protects against rapamycin-induced diabetic-like symptoms through activation of insulin/IGF signaling;Blättler;Cell Metab.,2012

4. Genomic binding of Pol III transcription machinery and relationship with TFIIS transcription factor distribution in mouse embryonic stem cells;Carrière;Nucleic Acids Res.,2012

5. The homeobox protein Prox1 is a negative modulator of ERRα/PGC-1α bioenergetic functions;Charest-Marcotte;Genes Dev.,2010

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