Absence of both MGME1 and POLG EXO abolishes mtDNA whereas absence of either creates unique mtDNA duplications
Author:
Funder
Research to Prevent Blindness
Muscular Dystrophy Association
Florida Department of Health
NEI
NINDS
US Army Research Office
Publisher
Elsevier BV
Reference34 articles.
1. Mammalian mitochondrial DNA replication and mechanisms of deletion formation;Falkenberg;Crit. Rev. Biochem. Mol. Biol.,2020
2. Mechanisms of replication and repair in mitochondrial DNA deletion formation;Fontana;Nucleic Acids Res.,2020
3. POLG-related disorders and their neurological manifestations;Rahman;Nat. Rev. Neurol.,2019
4. Mitochondrial DNA mutations, oxidative stress, and apoptosis in mammalian aging;Kujoth;Science,2005
5. Premature ageing in mice expressing defective mitochondrial DNA polymerase;Trifunovic;Nature,2004
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