Leishmania exploits host cAMP/EPAC/calcineurin signaling to induce an IL-33–mediated anti-inflammatory environment for the establishment of infection-Reference-Cited by-同舟云学术

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Leishmania exploits host cAMP/EPAC/calcineurin signaling to induce an IL-33–mediated anti-inflammatory environment for the establishment of infection

Published:2024-06 Issue:6 Volume:300 Page:107366
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Roy Souravi,Roy Shalini,Halder Satyajit,Jana Kuladip,Ukil Anindita

Publisher

Elsevier BV

Reference91 articles.

1. Involvement of IL-33 in the pathophysiology of systemic lupus erythematosus: review;Sarrand;Int. J. Mol. Sci.,2022

2. Interleukin-33 (IL-33): a nuclear cytokine from the IL-1 family;Cayrol;Immunol. Rev.,2018

3. Nuclear IL-33 is a transcriptional regulator of NF-κB p65 and induces endothelial cell activation;Choi;Biochem. Biophys. Res. Commun.,2012

4. IL-13 controls IL-33 activity through modulation of ST2;Zhang;J. Immunol.,2021

5. The ST2/IL-33 axis in immune cells during inflammatory diseases;Griesenauer;Front. Immunol.,2017

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