The ST2/IL-33 Axis in Immune Cells during Inflammatory Diseases-Reference-Cited by-同舟云学术

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The ST2/IL-33 Axis in Immune Cells during Inflammatory Diseases

Published:2017-04-24 Issue: Volume:8 Page:
ISSN:1664-3224
Container-title:Frontiers in Immunology
language:
Short-container-title:Front. Immunol.

Author:

Griesenauer Brad,Paczesny Sophie

Funder

National Cancer Institute

National Institute of Diabetes and Digestive and Kidney Diseases

Leukemia and Lymphoma Society

Publisher

Frontiers Media SA

Subject

Immunology,Immunology and Allergy

Reference199 articles.

1. Induction of a mitogen-responsive gene after expression of the Ha-ras oncogene in NIH 3T3 fibroblasts;Werenskiold;Mol Cell Biol,1989

2. A putative protein of a growth specific cDNA from BALB/c-3T3 cells is highly similar to the extracellular portion of mouse interleukin 1 receptor;Tominaga;FEBS Lett,1989

3. Soluble form of the ST2 gene product exhibits growth promoting activity in NIH-3T3 cells;Tominaga;Biochem Biophys Rep,2016

4. Regulation and function of T1/ST2 expression on CD4+ T cells: induction of type 2 cytokine production by T1/ST2 cross-linking;Meisel;J Immunol,2001

5. IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines;Schmitz;Immunity,2005

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