Bioactive lipid lysophosphatidic acid species are associated with disease progression in idiopathic pulmonary fibrosis
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Endocrinology,Biochemistry
Reference52 articles.
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2. An official ATS/ERS/JRS/ALAT clinical practice guideline: treatment of idiopathic pulmonary fibrosis. An update of the 2011 clinical practice guideline;Raghu;Am. J. Respir. Crit. Care Med.,2015
3. Acute exacerbation of idiopathic pulmonary fibrosis: shifting the paradigm;Ryerson;Eur. Respir. J.,2015
4. Acute exacerbation of idiopathic pulmonary fibrosis: who to treat, how to treat;Kulkarni;Curr. Pulmonol. Rep.,2019
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1. Lipid Deficiency Contributes to Impaired Alveolar Progenitor Cell Function in Aging and Idiopathic Pulmonary Fibrosis;American Journal of Respiratory Cell and Molecular Biology;2024-08
2. Lysophosphatidic acid receptor 1 inhibition: a potential treatment target for pulmonary fibrosis;European Respiratory Review;2024-04-30
3. Discovery of the Novel, Orally Active, and Selective LPA1 Receptor Antagonist ACT-1016-0707 as a Preclinical Candidate for the Treatment of Fibrotic Diseases;Journal of Medicinal Chemistry;2024-02-13
4. ISABELA Studies: Plasma Exposure and Target Engagement Do Not Explain the Lack of Efficacy of Ziritaxestat in Patients with Idiopathic Pulmonary Fibrosis;Clinical Pharmacology & Therapeutics;2024-01-05
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