Absence of DNA ligase IV protein in XR-1 cells: evidence for stabilization by XRCC4
Author:
Publisher
Elsevier BV
Subject
Genetics,Toxicology,Molecular Biology
Reference27 articles.
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3. Absence of a Ku-like DNA end binding activity in the xrs double strand break repair deficient mutant;Getts;J. Biol. Chem.,1994
4. Ku: product of the XRCC5 gene and it's role in DNA repair and V(D)J recombination;Taccioli;Science,1994
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1. Critical residues in the Ku70 von Willebrand A domain mediate Ku interaction with the LigIV-XRCC4 complex in non-homologous end-joining;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;2024-10
2. Quantitative proteome analysis of LAP1-deficient human fibroblasts: A pilot approach for predicting the signaling pathways deregulated in LAP1-associated diseases;Biochemistry and Biophysics Reports;2024-09
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4. The Mechanism, Regulation and Evolution of V(D)J Recombination;Molecular Biology of B Cells;2024
5. Androgen signaling stabilizes genomes to counteract senescence by promoting XRCC4 transcription;EMBO reports;2023-11-13
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