Interaction of FGF, Ihh/Pthlh, and BMP Signaling Integrates Chondrocyte Proliferation and Hypertrophic Differentiation-Reference-Cited by-同舟云学术

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Interaction of FGF, Ihh/Pthlh, and BMP Signaling Integrates Chondrocyte Proliferation and Hypertrophic Differentiation

Published:2002-09 Issue:3 Volume:3 Page:439-449
ISSN:1534-5807
Container-title:Developmental Cell
language:en
Short-container-title:Developmental Cell

Author:

Minina Eleonora,Kreschel Conny,Naski Michael C.,Ornitz David M.,Vortkamp Andrea

Publisher

Elsevier BV

Subject

Developmental Biology,Cell Biology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology

Reference46 articles.

1. A recurrent mutation in the tyrosine kinase domain of fibroblast growth factor receptor 3 causes hypochondroplasia;Bellus;Nat. Genet.,1995

2. Hedgehog and Bmp genes are coexpressed at many diverse sites of cell-cell interaction in the mouse embryo;Bitgood;Dev. Biol.,1995

3. Endogenous and ectopic expression of noggin suggests a conserved mechanism for regulation of BMP function during limb and somite patterning;Capdevila;Dev. Biol.,1998

4. Gly369Cys mutation in mouse FGFR3 causes achondroplasia by affecting both chondrogenesis and osteogenesis;Chen;J. Clin. Invest.,1999

5. A Ser(365)→Cys mutation of fibroblast growth factor receptor 3 in mouse downregulates Ihh/PTHrP signals and causes severe achondroplasia;Chen;Hum. Mol. Genet.,2001

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