Protein kinase C beta in postischemic brain mitochondria-Reference-Cited by-同舟云学术

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Protein kinase C beta in postischemic brain mitochondria

Published:2012-01 Issue:1 Volume:12 Page:138-143
ISSN:1567-7249
Container-title:Mitochondrion
language:en
Short-container-title:Mitochondrion

Author:

Kowalczyk Joanna E.,Kawalec Maria,Beręsewicz Małgorzata,Dębski Janusz,Dadlez Michał,Zabłocka Barbara

Funder

Polish Mitochondrial Network MitoNet.pl

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Molecular Medicine

Reference29 articles.

1. Regulation of intermediary metabolism by the PKC{delta} signalosome in mitochondria;Acin-Perez;FASEB J.,2010

2. Protein kinase Cepsilon interacts with and inhibits the permeability transition pore in cardiac mitochondria;Baines;Circ. Res.,2003

3. Identification of a voltage-gated potassium channel in gerbil hippocampal mitochondria;Bednarczyk;Biochem. Biophys. Res. Commun.,2010

4. Sevoflurane-induced cardioprotection depends on PKC-alpha activation via production of reactive oxygen species;Bouwman;Br. J. Anaesth.,2007

5. Cardioprotective mechanisms of PKC isozyme-selective activators and inhibitors in the treatment of ischemia–reperfusion injury;Budas;Pharmacol. Res.,2007

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1. Insulin-like Growth Factor II Prevents MPP+ and Glucocorticoid Mitochondrial-Oxidative and Neuronal Damage in Dopaminergic Neurons;Antioxidants;2021-12-24

2. Hippocampal Sector–Specific Metabolic Profiles Reflect Endogenous Strategy for Ischemia-Reperfusion Insult Resistance;Molecular Neurobiology;2020-11-22

3. cPKC beta II is significant to hypoxic preconditioning in mice cerebrum;Frontiers in Bioscience;2020

4. Gangliosides GM1 and GD1a normalize respiratory rates of rat brain mitochondria reduced by tert-butyl hydroperoxide;Journal of Evolutionary Biochemistry and Physiology;2017-05

5. Ischemia/Reperfusion-Induced Translocation of PKCβII to Mitochondria as an Important Mediator of a Protective Signaling Mechanism in an Ischemia-Resistant Region of the Hippocampus;Neurochemical Research;2017-04-12

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